Noopept (Omberacetam / GVS-111)

N-phenylacetyl-L-prolylglycine ethyl ester (synthetic dipeptide drug)
Evidence Level
Preliminary
4 Clinical Trials
5 Documented Benefits
1/5 Evidence Score

Noopept, international name omberacetam and lab code GVS-111, is a synthetic drug designed at a Moscow pharmacology institute as a dipeptide analog of piracetam. It is a registered medicine in Russia, sold there as 10 mg tablets for memory, attention and emotional complaints linked to head injury, vascular brain disease and asthenic states. It is not a dietary supplement: FDA warning letters have told US sellers that Noopept products are unapproved new drugs, and it is not authorised for human use in the EU or Australia. PubMed indexes no placebo-controlled human trial. The clinical data are small Russian studies in patients, and no controlled trial tests memory or focus in healthy people. When US researchers tested cognitive supplements, omberacetam turned up at up to four times the medical dose, and most declared drug amounts were wrong.

Studied Dose Russian label: 10 mg twice daily (20 mg/day), up to 30 mg/day, in 1.5 to 3 month courses; the stroke study used 20 mg/day
Active Compound Omberacetam (N-phenylacetyl-L-prolylglycine ethyl ester, GVS-111)

Benefits

Cognition after stroke: small Russian studies only

In an open study of stroke patients with mild cognitive impairment, 20 mg a day for two months improved a cognitive screening score and verbal fluency more than in a comparison group, with no blinding and no placebo described. A study against piracetam in vascular and post-traumatic cognitive disorders is often cited, but it has no indexed abstract, so its size, design and results cannot be checked.

Memory and focus in healthy people: no controlled trial

PubMed indexes no controlled trial of Noopept for memory, focus or learning in healthy adults. Its reputation as a tiny dose that sharpens thinking comes from animal work, user anecdotes and Russian labeling written for patients. The few Russian studies in healthy people looked at adapting to heat, cold and thin air in young volunteers, or at work capacity when older teachers added self-massage to Noopept; neither was a memory trial.

Brain cell protection in lab and animal models

In cultured human cortical neurons, Noopept limited cell death and free radical damage caused by hydrogen peroxide, and in rats it raised hippocampal messenger RNA for the growth factors NGF and BDNF. Every mechanism paper cited on this page includes scientists from the institute that created the drug, and none of it shows protection of the human brain.

Regulatory status: an unapproved drug, not a supplement

FDA warning letters have told US sellers that Noopept products are unapproved new drugs rather than dietary supplements, and US law does not let unapproved drugs enter supplements as new ingredients. European medicines laboratories report it is not authorised for human use in the EU or Australia. In Russia it is a registered medicine sold as 10 mg tablets.

What is actually in the bottle is unpredictable

When US researchers tested ten cognitive supplements, every one contained omberacetam, at about 5 to 41 mg per serving against a usual medical dose of 10 mg. Nine of twelve declared drug amounts were wrong, and two products held undeclared unapproved drugs such as phenibut or picamilon. Medicines control labs in Europe and Australia have also intercepted bulk Noopept raw material of up to 100% purity.

Mechanism of action

1

Prodrug of the brain peptide cyclo-prolylglycine

In rats, Noopept itself was undetectable in brain an hour after injection, while cyclo-prolylglycine, a small cyclic peptide the brain makes on its own, rose about 2.5-fold. Plasma and brain enzymes formed it from Noopept in vitro, so the developers describe the drug as a prodrug of that peptide.

2

Growth factor gene expression in the hippocampus

Single and 28-day dosing in rats raised hippocampal messenger RNA for NGF and BDNF, and the effect did not fade with repeated dosing. In the cortex a single dose lowered expression and chronic dosing raised BDNF only slightly. These are gene expression changes in rats, not memory gains in people.

3

Alpha7 nicotinic receptor signaling

In rat hippocampal slices, Noopept increased the firing of inhibitory interneurons, and drugs that block alpha7 nicotinic acetylcholine receptors almost abolished the effect. That ties it to cholinergic signaling involved in attention and memory, but only at the level of brain slice recordings.

4

Antioxidant protection in cultured neurons

In human cortical neurons exposed to hydrogen peroxide, Noopept improved survival in a dose-dependent way and reduced free radical build-up and lipid peroxidation, outperforming piracetam and vitamin E in the same experiments. Concentrations in a culture dish do not show what an oral dose achieves in the brain.

Clinical trials

1
Noopept After Stroke - Open Prospective Study
PubMed

Open prospective Russian study of Noopept in stroke patients with mild cognitive impairment, compared with a control group (Amelin AV, Iliukhina AIu, Shmonin AA 2011, Zh Nevrol Psikhiatr Im S S Korsakova 111(10 Pt 1):44-6, PMID 22500312).

60 stroke patients; the abstract says they were treated with Noopept during 12 months, yet the abstract reports a Noopept dose of 20 mg a day for 2 months and does not say how patients were split between groups.

MMSE scores and verbal association tasks improved significantly after 2 months in the Noopept group compared with controls, and the global efficacy rating showed mild improvement versus no change in controls. The authors described safety as high but the abstract gives no adverse event counts. The design was open, with no blinding and no placebo described, and only a short Russian-language abstract is indexed.

2
Noopept Versus Piracetam - Comparative Study
PubMed

Comparative clinical study of Noopept and piracetam from the clinical psychopharmacology laboratory of the Zakusov Institute of Pharmacology in Moscow, the institute whose scientists designed Noopept (Neznamov GG, Teleshova ES 2009, Neurosci Behav Physiol 39(3):311-21, PMID 19234797).

Patients with mild cognitive disorders caused by cerebrovascular disease or brain injury.

This head-to-head study is frequently cited for Noopept, yet PubMed indexes both the English translation and the Russian original without an abstract, so the number of patients, the blinding and the results cannot be checked from the indexed record. Its title describes a comparison of two drugs, and a head-to-head comparison on its own cannot show whether either one beat no treatment.

3
Unapproved Drugs in US Cognitive Supplements
PubMed

Laboratory analysis of over-the-counter supplements labeled as containing racetam-type drugs (Cohen PA, Avula B, Wang YH, Zakharevich I, Khan I 2021, Neurol Clin Pract 11(3):e303-e307, PMID 34484905). A product-quality study, not a clinical trial.

Ten cognitive enhancement supplements bought online in the US; no human participants.

Omberacetam was present in all 10 products at 5.1 to 40.6 mg per serving, against a typical pharmacologic dose of 10 mg. Of 12 declared drug quantities, 9 were inaccurate, ranging from 0% to 135% of the label. Two products held undeclared aniracetam, phenibut or picamilon, and one product combined four unapproved drugs.

4
Illicit Nootropics in Europe and Australia - Market Surveillance
PubMed

Market surveillance study by 12 official medicines control laboratories in Europe and Australia (Vanhee C, Deconinck E, George M, et al. 2025, J Xenobiot 15(3):88, PMID 40558871).

159 samples of suspected smart drugs and nootropics collected from January 2020 to September 2024, most sold as dietary supplements or medicines and most from the illegal market.

Noopept was the most commonly detected racetam, often seized as bulk raw material of up to 100% purity, and one liquid held 20 mg/mL, twice the typical oral dose used in Russia. The laboratories note it is not authorised for human use in the EU or Australia and warn that consumers buying such products as supplements may not know they are taking a drug.

Side effects and drug interactions

Common Potential side effects

The Russian label lists allergic reactions as the main possible side effect.
Blood pressure can rise in people with hypertension, according to the Russian label.
The Russian label advises taking the last dose no later than 6 pm.
Contraindicated in pregnancy, breastfeeding, under age 18, and severe liver or kidney disease per the Russian label.
Supplement products have held up to four times the medical dose, sometimes alongside undeclared drugs such as phenibut.
Bulk powder is dosed in milligrams, so scooping errors can multiply the intended dose.
Safety has been described only for supervised courses of weeks to a few months; long-term self-dosing has not been studied.

Important Drug interactions

No human interaction studies were found; the Russian label reports no interaction found with alcohol, sleeping pills or blood pressure drugs, which reflects limited testing rather than proven safety.
Blood pressure medicines: the label warns that blood pressure may rise in people with hypertension, so monitor readings.
Valproate and other seizure medicines: in mice, five weeks of Noopept strengthened valproate's anticonvulsant effect; do not add it without the prescribing neurologist.
Warfarin, heparin and antiplatelet drugs: animal and test-tube work reported anticoagulant and fibrinolytic activity; the combination is untested.
Cholinesterase inhibitors such as donepezil: overlapping cholinergic activity in lab work; the combination is untested.
Other racetams, phenibut, picamilon and stimulants: often stacked, never tested together, and supplement products have contained them undeclared.

Frequently asked questions about Noopept (Omberacetam / GVS-111)

What is Noopept?

Noopept, international name omberacetam and lab code GVS-111, is a synthetic drug designed at a Moscow pharmacology institute as a dipeptide analog of piracetam. It is a registered medicine in Russia, sold there as 10 mg tablets for memory, attention and emotional complaints linked to head injury, vascular brain diseas…

What is Noopept used for?

Noopept is researched primarily for Cognitive. In an open study of stroke patients with mild cognitive impairment, 20 mg a day for two months improved a cognitive screening score and verbal fluency more than in a comparison group, with no blinding and no placebo described.

What is the recommended dosage of Noopept?

The clinically studied dose is Russian label: 10 mg twice daily (20 mg/day), up to 30 mg/day, in 1.5 to 3 month courses; the stroke study used 20 mg/day Always follow the product label and check with a healthcare provider for personal advice.

Is Noopept safe, and does it have side effects?

For most healthy adults, Noopept is well tolerated at studied doses. Reported effects can include: The Russian label lists allergic reactions as the main possible side effect. Blood pressure can rise in people with hypertension, according to the Russian label. It may also interact with some medications. Noopept is not right for everyone, so check with a healthcare provider first if you are pregnant or breastfeeding, have a medical condition, or take prescription medication.

Does Noopept interact with any medications?

Possible interactions include: No human interaction studies were found; the Russian label reports no interaction found with alcohol, sleeping pills or blood pressure drugs, which reflects limited testing rather than proven safety. If you take prescription medication, check with a pharmacist or doctor before using it.

How strong is the scientific evidence for Noopept?

NutraSmarts rates the evidence for Noopept as Preliminary (1 out of 5). It is backed by 4 clinical trials and 8 cited references summarized on this page. A higher rating reflects more, larger, and better-designed human studies.

References(8 citations)

Evidence ratings on NutraSmarts are based on the totality of human clinical research, with emphasis on randomized controlled trials, meta-analyses, and systematic reviews. The references below directly support claims made throughout this page.

  1. Cohen PA, Avula B, Wang YH, Zakharevich I, Khan I. Five Unapproved Drugs Found in Cognitive Enhancement Supplements. Neurol Clin Pract. 2021;11(3):e303-e307..PubMedUsed to support: Laboratory analysis of 10 US cognitive supplements: omberacetam was present in all 10 at 5.1 to 40.6 mg per serving versus a typical 10 mg pharmacologic dose, 9 of 12 declared drug quantities were inaccurate, and undeclared aniracetam, phenibut and picamilon turned up in two products. It also records that FDA has sent warning letters to sellers of omberacetam and that US law does not permit unapproved drugs as new supplement ingredients.
  2. Vanhee C, Deconinck E, George M, Hansen A, Hackl A, Wollein U, El-Atma O, Beerbaum N, Aureli F, Borioni A, Poplawska M, Blazewicz A, Roschel K, Marson C, Mendoza Barrios M, Hakkarainen B, Blomgren A, Bakker-'t Hart I, Miquel M. The Occurrence of Illicit Smart Drugs or Nootropics in Europe and Australia and Their Associated Dangers: Results from a Market Surveillance Study by 12 Official Medicines Control Laboratories. J Xenobiot. 2025;15(3):88..PubMedUsed to support: Official medicines control laboratories analysed 159 samples collected from 2020 to 2024. Noopept was the most commonly detected racetam, often seized as bulk raw material of up to 100% purity, and the authors state it is not authorised for human use in the EU or Australia.
  3. Amelin AV, Iliukhina AIu, Shmonin AA. [Noopept in the treatment of mild cognitive impairment in patients with stroke]. Zh Nevrol Psikhiatr Im S S Korsakova. 2011;111(10 Pt 1):44-6..PubMedUsed to support: Open prospective Russian study in 60 stroke patients in which 20 mg a day for 2 months improved MMSE and verbal association scores compared with controls. Open design with no blinding and no placebo described; the basis for the post-stroke cognitive claim on this page.
  4. Neznamov GG, Teleshova ES. Comparative studies of Noopept and piracetam in the treatment of patients with mild cognitive disorders in organic brain diseases of vascular and traumatic origin. Neurosci Behav Physiol. 2009;39(3):311-21..PubMedUsed to support: The comparative Noopept versus piracetam study in mild cognitive disorders of vascular and traumatic origin, from the clinical psychopharmacology laboratory of the Zakusov Institute. PubMed carries no abstract, so its size, design and results cannot be verified from the indexed record.
  5. Gudasheva TA, Boyko SS, Ostrovskaya RU, Voronina TA, Akparov VK, Trofimov SS, Rozantsev GG, Skoldinov AP, Zherdev VP, Seredenin SB. The major metabolite of dipeptide piracetam analogue GVS-111 in rat brain and its similarity to endogenous neuropeptide cyclo-L-prolylglycine. Eur J Drug Metab Pharmacokinet. 1997;22(3):245-52..PubMedUsed to support: Rat study in which Noopept was undetectable in brain an hour after injection while cyclo-prolylglycine rose 2.5-fold, with conversion by plasma and brain enzymes shown in vitro; the basis for the prodrug mechanism on this page.
  6. Ostrovskaya RU, Gudasheva TA, Zaplina AP, Vahitova JV, Salimgareeva MH, Jamidanov RS, Seredenin SB. Noopept stimulates the expression of NGF and BDNF in rat hippocampus. Bull Exp Biol Med. 2008;146(3):334-7..PubMedUsed to support: Rat study in which single and 28-day Noopept dosing raised hippocampal NGF and BDNF mRNA without tolerance, while cortical expression fell after a single dose. Animal gene expression data only, from the drug's developers.
  7. Kondratenko RV, Povarov IS, Kolbaev SN, Derevyagin VI, Ostrovskaya RU, Gudasheva TA, Sharonova IN, Skrebitsky VG. Effect of nootropic dipeptide noopept on CA1 pyramidal neurons involves α7AChRs on interneurons in hippocampal slices from rat. Neurosci Lett. 2022;790:136898..PubMedUsed to support: Rat hippocampal slice recordings in which Noopept's excitation of inhibitory interneurons, and the resulting rise in spontaneous inhibitory current frequency in pyramidal cells, was almost abolished by alpha7 nicotinic receptor antagonists.
  8. Pelsman A, Hoyo-Vadillo C, Gudasheva TA, Seredenin SB, Ostrovskaya RU, Busciglio J. GVS-111 prevents oxidative damage and apoptosis in normal and Down's syndrome human cortical neurons. Int J Dev Neurosci. 2003;21(3):117-24..PubMedUsed to support: Cell-culture study in normal and Down syndrome human cortical neurons: Noopept improved survival after hydrogen peroxide, reduced free radicals and lipid peroxidation, and outperformed piracetam and vitamin E in vitro. Co-authored by the drug's developers; no human participants.